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Doctor Finds Natural Ways to Reverse and Prevent Alzheimer's

Doctor Finds Natural Ways to Reverse and Prevent Alzheimer's Read Transcript


LORIE JOHNSON: After 30 years of research,

Dr. Dale Bredesen says we now know

why we get Alzheimer's, and therefore

how we can reverse it and even prevent it.

He's proven this in trials on people

with mild to moderate declines in their memory and ability

to think.

As he explains in his new book, "The End of Alzheimer's,"

the key is understanding that Alzheimer's can

have up to 36 different causes.

Just like a roof with 36 holes in it

can only work well if all 36 are repaired,

all 36 potential causes of Alzheimer's must

be addressed for a person's brain to function properly.

The causes, he says, include genetics, exposure to metals,

toxins, a poor diet, and more.

Dr. Bredesen says each of us can see

where we are when it comes to all 36 Alzheimer's risk factors

by taking a test call a cognoscopy.

The results from that test are plugged into a computer

algorithm to determine your own individualized treatment,

based on your own strengths and weaknesses.

Dr. Bredesen recommends everyone get a cognoscopy beginning

at age 45 to prevent all timers from taking hold.

Lorie Johnson, CBN News.

Thanks, Lorie.

The book is called "The End of Alzheimer's," and Dr. Dale

Bredesen is here with us now.

Doctor, this is a breakthrough.

You researched according, to your book--

is there any literature anywhere that says we have a cure

for Alzheimer's.

No, and we can't call this a cure yet.

Because you really need to look at an autopsy to say,

you know, the entire process has gone.

INTERVIEWER: OK.

We can't say that yet.

What we can say is, for the first time

we can reverse the symptoms.

So we can make people better, and we

have now over 1,000 people who have been on the protocol.

And the key is to look at what's actually driving it.

And just as you wouldn't say, well, you

can ignore nine commandments and just follow one,

if we find the same thing.

When you look, this is a complex, chronic illness.

And if you remember what happened with HIV,

it took three drugs.

One drug doesn't do very well.

INTERVIEWER: All right.

Alzheimer's is much more complicated than HIV.

So we find many different things that have to be addressed.

Nobody has dared to come out with a so-called protocol

to end it.

You're the first one.

Am I right in that?

That's correct.

So in 2014, we published the first peer

reviewed paper that showed--

this was initially just 10 people.

And I have to say, when we got the first call

from the first person who said she was back at work

and things were great, I was really surprised and excited.

Because we'd been working on this for so many years.

You see it again and again.

What is Alzheimer's?

You know, we had President Reagan, who

was beloved by so many, had this disease

and he wrote that thing, I'm going into the darkness.

And what happens when somebody gets Alzheimer's?

This is a great question, Pat.

And you know, in the book we talk about someone who actually

descended and came back.

And so she wrote about what it actually

felt like to be descending into Alzheimer's, and then

what it felt like to come back and to be well again

and to be healthy again.

So this is a disease that has a number

of different contributors.

As Lorie mentioned, we identified, initially,

36 things.

But these all contribute through the same network

to the same ultimate change.

Well, people thought, OK, we found amyloid plaques,

and yipee, we have an answer.

We didn't have an answer for that, did we?

That's exactly right.

Here's the surprise, Pat.

Amyloid is actually a protective response,

which hadn't been appreciated before,

to three fundamentally different insults that happened

to your brain, chronic inflammation,

a reduction of trophic support-- those are nutrients

and hormones and factors and proteins in your brain that

support your neurons--

and then exposure to toxins.

If you're exposed to certain toxins,

as Lorie had mentioned, from mycotoxins, from molds,

or things like mercury, your brain actually

makes this amyloid that we pick up

as the disease as a protective response to that.

So it's a very different look than what we originally

thought.

Let's go up against the those insults.

The trophic, for example--

what exactly is that?

So your brain needs dozens and dozens of factors.

You have a very complicated computer inside your skull,

and it needs not only things like vitamin D and vitamin B12,

it needs things like estradiol and progesterone

and testosterone, nerve growth factor,

which is a protein made by your brain, brain

derived neurotrophic factor.

There are dozens and dozens of these factors.

And what happens is, as you get a little older,

you can decrease the production of those

so that it changes a fundamental balance.

You have a beautiful balance throughout most

of your life in your brain's structure,

and a change in that balance could ultimately

lead to this pulling back that we refer

to as Alzheimer's disease.

What is it that would be the cause?

What kind of insult would take place in the brain that would

tend toward Alzheimer's?

A very good point.

There are dozens and dozens of things.

So for example, if you have chronic exposure to pathogens,

such as Lyme disease, that's one thing,

your body tries to fight the Lyme disease by making

the amyloid that you then pick up in the brain as Alzheimer's.

If you have mold in your home, surprisingly,

it turns out that the molds actually

make specific toxins called mycotoxins.

And there are several of these, tricothecenes and ochratoxin,

and things like this.

You make this, again, to bind up that toxin.

You're trying to protect yourself from this.

But in so doing, you are also downsizing.

So is this mold--

if we live in a wet area here down on the ocean,

mold will tend towards Alzheimer's if you live in--

So in some people-- that's a great point.

So some people are very good at avoiding it

and very good at excreting it.

So we're all detoxing ourselves from these various things.

And most of us successfully do that.

But for people who have some genetic backgrounds--

and there's something called HLA DR-DQ--

it's a genetic marker that can actually tell you

whether you have sensitivity.

So we recommend people get this evaluation

we call a cognoscopy.

Anyone over 45, get an evaluation

to see where do you stand.

Do you have, in fact, an environment that's a problem?

Do you have insulin resistance, which is one

of the very common problems.

If you go out and eat a bunch of processed food and a bunch

of sugar and things like that, you

develop what's called insulin resistance.

Your brain no longer responds to the insulin in the way

that it should.

And that is one of the important contributors

to Alzheimer's disease.

Looks like inflammation is one of the chief causes of all

of pathology that we have.

You want to talk about that?

Absolutely.

So inflammation, chronic inflammation, as you know,

for heart disease it's a problem.

And it's also a problem for Alzheimer's.

And it leads to what we call Type 1 Alzheimer's.

So there are these different subtypes that we discovered.

And one of them is this inflammatory type.

And you know, the ayuvedic physicians,

thousands of years ago, knew about this.

And they called this pitta.

Pitta, in dementia-- now, they didn't call it

Alzheimer's of course--

but they knew about dementia, and they called that type

pitta, which is "hot."

There was a story about a group,

I believe it was in Colombia, that

had a genetic predisposition.

How extensive is that?

That's exactly right.

It's a very uncommon cause worldwide,

and very uncommon cause in America.

So fewer than 5% of people will have this genetically

predetermined Alzheimer's.

And that particular one that you mentioned

is a gene called Presenilin-1, or PS1.

And it is a cause of familial, but most 95% of Alzheimer's is

so-called sporadic Alzheimer's.

It is associated with risk from a gene called APOE4.

So good idea to check out your APO status.

And people used to say, I don't want to know,

because I don't-- there's nothing you can do.

That's wrong.

There is a tremendous amount that people can do.

36 factors, though-- in your book,

people have got to be alert to these all 36 though,

don't they?

Well, so the thing is, you can now go in and we've trained now

450 practitioners from seven different countries and all

over the United States, so that you can go--

including some for some practitioners from Virginia--

and you can go and you can find the specific causes.

You can look and see what your status is,

and then you can work with a health

coach who will help you to get on the optimal program.

And we've developed, as Laurie said, a computer based program

that tells you what are the most important things for you,

and what is your optimal program.

Give us a couple of examples of people who have had--

they've sunk into the deep night of Alzheimer's and they've come

back.

It's absolutely striking, Pat.

And I have to say, I thought I was

going to spend my whole career with transgenic mice.

And so back in 2012, we had our first patient come through.

And I talk in the book, for example,

about one person, Edward, who was

unable to remember the people he had lunch with, for example.

He had quantitative neurocognitive testing

that showed that he had sunk down

to the third percentile for his age.

He had a PET scan that showed classic Alzheimer's disease.

He also had the risk gene, which is the APOE4.

So he had all of those, and got on the program,

and within six months, improved.

And finally, he went back for more quantitative testing

and he'd gone from the third percentile

to the 84th percentile, so doing much better than average.

He was literally closing up shop.

He's now opened a new office and has three offices.

INTERVIEWER: That's good.

Is now four years on the program

and doing absolutely splendidly.

Well, the program, primarily you're

dealing with inflammation.

You're dealing with the mold, you're dealing with toxins,

and you're supplying the nutrients that people

need to nourish their brain.

Is that essentially it?

The bottom line here is that people do not

get this disease for no reason.

And we keep being told it's mysterious.

There's nothing you can do about it.

It's not mysterious.

There are factors that contribute to this, just

as there are to heart disease--

no different.

And so you look at all these things,

and you can actually see, for each person,

what is driving the problem.

And then what you want to do is attack all those things.

And you want to optimize.

You change the balance, literally,

between the memory side and the forgetting side.

Peer review-- are your associates going along with

what you've found?

It's a huge breakthrough, in my opinion.

Thank you, very much.

So we've had a lot of positive feedback,

and we've had over 5,000 e-mails about just the first paper.

We've published a few more.

There are now over 1,000 people on this.

There was an article in "The London Times"

today about our work.

And, of course, you can imagine the experts are skeptical.

That's OK.

As we have more and more people, the skepticism will go away.

If your work is taken out of the equation, there is nothing,

am I correct?

That is correct.

There is no alternative right now.

The drugs do not help very much at all.

And our argument is, if you're going to have a drug, fine.

But do it on the background of the entire program.

So you're talking about removing the causes

of inflammation, removing the toxins that are in the system

that may be affecting people, giving them the tropic material

that they need to build up their brains, and that's it--

it starts working?

It's getting at the root causes

and optimizing these many different factors.

That's when we begin to see this turn around.

And so here's a challenge for all of us.

Let's all work together to reduce

the global burden of dementia.

Because this is a major problem throughout the world.

If somebody gets your book, do they have the clue?

I mean, do they have to get one of these tests?

So the book goes through all the different things.

It goes through all the different tests,

the scientific background for this,

and it's written for everyone.

So it goes through all the different tests you can get,

where you can get them, what they mean,

how to interpret them, and then what is the program.

And it even has specific programs

that have worked for specific people,

and people who are talked about their own programs.

You're talking about 75 million people

at risk of this stuff.

I mean, this is huge.

This is now the third leading cause of death in the US,

and the number one cause of death in the UK, past heart

disease and it passed cancer.

In the UK number one?

Dementia is the number one cause of death in the UK.

So this is a big problem.

The tragedy that comes upon a family when

their loved one is suddenly out of it and then facing death.

Because most of them die if they've got this, don't they?

Yeah, as they say, everyone knows

someone who's survived cancer.

No one knows someone who's survived Alzheimer's.

So these are the first.

Doctor, god bless you.

I hope people listen.

Ladies and gentlemen, the book is called "The End

of Alzheimer's."

It's available wherever books are sold.

Dr. Dale Bredesen is--

well, it's monumental research that he's done.

Nobody, in my opinion, has come up with any kind of a solution

to Alzheimer's.

When people have it-- and I've had some dear friends

that go into that thing, and it is just beyond belief

the tragedy in people's homes.

So let's do what we can.

Doctor, thank you.

Thank you very much, Pat.

Thanks for having me on.

All right.

Dr. Dale Bredesen, ladies and gentlemen.

The book again is called "The End of Alzheimer's."

This is not an easy read, I might add.

To say this is a fun book would be a lie.

I've got to say, it is very complex.

And this 36 step is complex, but so is Alzheimer's.

God bless you.

Thank you.

Thank you very much, Pat.

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